Module number 983




Database revision : gnsdb28.10
Date : Tue Feb 25 17:20:49 2003
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mKRE23:Killer toxin REsistant,,Null mutant is K1 killer toxin resis\ntent\n Cond626:DY1457_(wild_type)_3_mM_vs._61_nM_zinc_y13-121\n mYBR113W:Unknown ,, Unknown\n mYOL083W:Unknown ,, Unknown\n mTFS1:(putative) lipid binding protein; supressor of a cdc25 mutat\nion,lipid binding protein (putative) , supressor of a cdc25 \nmutation,Null mutant is viable.\n mGDB1:Glycogen debranching enzyme; the enzyme that debranches the \nglycogen having a glucanotranferase + 1-6amyloglucosidase ac\ntivity,,Null mutant is viable but unable to degrade glycogen\n.\n mMKK2:Member of MAP kinase pathway involving PKC1, BCK1, and SLT2.\n Shows functional redundancy with MKK1,protein kinase,Null m\nutant is viable and shows no obvious phenotypes; mkk1 mkk2 d\nouble mutant is caffeine-sensitive and shows a temperature-s\nensitive cell lysis defect remediated by osmotic stabilizers\n mYFR026C:Unknown ,, Unknown\n mYDR271C:Unknown ,, Unknown\n mYCL042W:Unknown ,, Unknown\n mCYM1:Unknown ,, Unknown\n mYOR192C:Unknown ,, Unknown\n mYOL015W:Unknown ,, Unknown\n mYER188W:Unknown ,, Unknown\n mUBP16:putative deubiquitinating enzyme,deubiquitinating enzyme (pu\ntative),\n mSDP1:Unknown ,, Unknown\n mYEL015W:Unknown ,, Unknown\n mECM4:ExtraCellular Mutant,,A Tn3 insertion into this gene causes \nhypersensitivity to the cell surface polymer perturbing agen\nt calcofluor white.\n mPHR1:photolyase,photolyase,photoreactivation repair deficient\n mPOX1:fatty-acyl coenzyme A oxidase,fatty-acyl coenzyme A oxidase,\nNull mutant is viable, exhibits diminished ability to use ol\neic acid as a carbon source\n mYBL064C:Unknown ,, Unknown\n mPEX18:Peroxin; Pex18p and Pex21p are partially functionally redund\nant.,peroxin,Null mutant is viable but has reduced growth on\n oleate, partial impairment of peroxisome biogenesis\n mMF(ALPHA)2:alpha mating factor,alpha mating factor,Null mutant is viabl\ne.\n mYGL198W:Unknown ,, Unknown\n mGPM2:Similar to GPM1 (phosphoglycerate mutase); converts 3-phosph\noglycerate to 2-phosphoglycerate in glycolysis,,Null mutant \nis viable, gpm2 gpm3 double deletion mutants exhibit no synt\nhetic phenotypes\n mYLR125W:Unknown ,, Unknown\n mCIS1:Involved in microtubule assembly,,\n mYHR140W:Unknown ,, Unknown\n COMP.:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mCRS5:Metallothionein-like protein,metallothionein-like protein,Nu\nll mutant is viable, exhibits increased sensitivity to coppe\nr toxicity\n mNYV1:Synaptobrevin (v-SNARE) homolog involved in vacuolar vesicle\n fusion,vacuolar v-SNARE,Null mutant is viable\n mYGK3:Unknown ,, Unknown\n mMGT1:6-O-methylguanine-DNA methylase,6-O-methylguanine-DNA methyl\nase,Null mutant is viable, sensitive to alkylation induced k\nilling and mutation\n mYHR210C:Unknown ,, Unknown\n mYOL085C:Unknown ,, Unknown\n mYGR053C:Unknown ,, Unknown\n mPAI3:Cytoplasmic inhibitor of proteinase Pep4p,inhibitor of prote\ninase Pep4p,Null mutant is viable but shows increased rate o\nf protein degradation\n mYKR049C:Unknown ,, Unknown\n mYDL124W:Unknown ,, Unknown\n mYHL008C:Unknown ,, Unknown\n TorRama.Series0:Partitioning the transcriptional program induced by rapamyci\nn among the effectors of the Tor proteins. Curr Biol. 2000 D\nec 14-28;10(24):1574-81\n mYKL031W:Unknown ,, Unknown\n mHSP26:heat shock protein 26,heat shock protein 26,Null mutant is v\niable; hsp26 hsp42 double deletion mutants are viable\n mYDR269C:Unknown ,, Unknown\n mYPR150W:Unknown ,, Unknown\n mICY2:Interacting with the cytoskeleton,,\n mAPG1:Required for autophagy,protein kinase,Defective in autophagy\n; loses viability more rapidly than wild type during nitroge\nn starvation; defective in vacuolar protein degradation duri\nng nitrogen starvation; defective in sporulation\n mPUS2:pseudouridine synthase 2,pseudouridine synthase,Null mutant \nis viable\n Cond871:yhe710-ss\n mYOR364W:Unknown ,, Unknown\n mYJR038C:Unknown ,, Unknown\n mYPR151C:Unknown ,, Unknown\n mMDG1:multicopy suppressor of bem1 mutation, may be involved in G-\nprotein mediated signal transduction; binds cruciform DNA,,N\null mutant is viable. Deletion of MDG1 causes sterility in c\nells in which the wild-type G beta has been replaced by part\nly defective G beta derivatives\n mNDE2:Unknown ,, Unknown\n mOM45:45-kDa mitochondrial outer membrane protein,45 kDa mitochond\nrial outer membrane protein,Null mutant is viable and shows \nnormal growth, viability, mitochondrial function and mitocho\nndrial protein import\n mLSC2:beta subunit of succinyl-CoA ligase (synthetase; ATP-forming\n), a mitochondrial enzyme of the TCA cycle,,Null mutant is v\niable but grows slowly on minimal glycerol or pyruvate; muta\nnt suppresses idh2 null mutants for growth on glycerol\n mALD4:Glucose repressed. Utilizes NADP+ or NAD+ as a coenzyme equa\nlly well. (sold by SIGMA under the catalogue number A5550, a\nccording to A. Blomberg).,aldehyde dehydrogenase,\n mYCR061W:Unknown ,, Unknown\n mHPA2:Histone and other Protein Acetyltransferase; Has sequence ho\nmology to known HATs and NATs,histone acetyltransferase,Null\n mutant is viable and does not show any detectable phenotype\n mYBR075W:Unknown ,, Unknown\n mTRX2:thioredoxin,thioredoxin,Null mutant is viable; trx1-trx2 dou\nble mutant shows prolonged S phase, shortened G(sub)1 and me\nthionine auxotrophy\n mYGP1:may be involved in cellular adaptations prior to stationary \nphase,gp37, a glycoprotein synthesized in response to nutrie\nnt limitation which is homologous to the sporulation-specifi\nc SPS100 gene,\n mGPG1:Unknown ,, Unknown\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mCVT9:Oligomeric, coiled-coil, peripheral membrane protein,Oligome\nric, coiled-coil, peripheral membrane protein required for s\ntable binding of precursor API to its target membrane.,cvt9 \nis defective in maturation of the vacuolar protein, aminopep\ntidase I and exhibits minor defects in autophagy , cvt9 is d\nefective in vacuolar delivery of aminopeptidase I and peroxi\nsome degradation but is not needed for macroautophagy. The n\null mutant is viable and is relatively starvation-insensitiv\ne.\n mYGL146C:Unknown ,, Unknown\n mYHR087W:Unknown ,, Unknown\n mYBR210W:Unknown ,, Unknown\n mZIP1:Synaptonemal complex protein, component of the central eleme\nnt,,Null mutant is viable and shows defects in meiosis\n mRNY1:RiboNuclease from Yeast,ribonuclease, T2 family,\n mNGL3:DNase/RNase (putative); CCR4 C-terminal homolog; displays ho\nmology to drosophila Angel gene; homolog to ngl1 and ngl2,DN\nase (putative) , RNase (putative),\n mPUT4:putative proline-specific permease,proline-specific permease\n (putative),inability to use proline as a nitrogen source\n mYHR189W:Unknown ,, Unknown\n mYNL285W:Unknown ,, Unknown\n mYOL047C:Unknown ,, Unknown\n mAUT4:Autophagy gene essential for breakdown of autophagic vesicle\ns in the vacuole,,Null mutant is viable, but exhibits defect\ns in lysis of autophagic vesicles after delivery to the vacu\nole; vesicles accumulate in the vacuole in the absence of PM\nSF; maturation of the vacuolar protein, aminopeptidase I is \nunaffected in aut4\n mOSM1:osmotic growth protein,osmotic growth protein,Null mutant is\n viable, sensitive to hypertonic medium\n mYJL163C:Unknown ,, Unknown\n mYGR051C:Unknown ,, Unknown\n mYMR251W:Unknown ,, Unknown\n zap1.Series0:Genome-wide characterization of the Zap1p zinc-responsive re\ngulon in yeast. Proc Natl Acad Sci U S A. 2000 Jul 5;97(14)\n:7957-62.\n Cond627:DY1457_(wild_type)_3_mM_vs._76_nM_zinc_y12-122\n mPHM7:Hypothetical ORF,,transcription is regulated by PHO system\n mYJR003C:Unknown ,, Unknown\n mYLR415C:Unknown ,, Unknown\n mERV29:ER Vesicle protein of 29 kDa (apparent MW),ER-Golgi transpor\nt vesicle protein,Null mutant is viable.\n mYIL025C:Unknown ,, Unknown\n mSPR1:Sporulation regulated genes,exo-1,3-beta-glucanase, sporulat\nion-specific,Fail to hydrolyze p-nitrophenyl-beta-D-glucanas\ne or laminarin; mutant spores exhibit reduced thermoresistan\nce\n Cond652:wt_plus_gamma_20_min\n mMGT1 mGPG1
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Computational Genomics Lab, Tel-Aviv uniresity