Module number 831




Database revision : gnsdb28.10
Date : Tue Feb 25 17:07:11 2003
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mYMR316C-A:Unknown ,, Unknown\n Cond101:mrpl33\n Cond217:yer050c\n mYLR346C:Unknown ,, Unknown\n Cond3:aep2\n mYOL083W:Unknown ,, Unknown\n mYOR152C:Unknown ,, Unknown\n mPDR15:similar to Pdr5p and Pdr10p,multidrug resistance transporter\n (putative),\n mPDR16:involved in pleiotropic drug resistance by controlling lipid\ns in various cellular compartments; positively regulated by \nPDR1,Pdr17p homolog , Sec14p homolog,Null mutant is viable, \nexhibits hypersensitivity to azole inhibitors of ergosterol \nbiosynthesis, alterations in sterol composition of the plasm\na membrane; pdr16 pdr17 double deletion mutants exhibit addi\ntive exacerbated phenotypes\n mTYE7:may be involved in glycolytic gene expression,33 kDa , serin\ne-rich protein, is a potential member of the bHLH/leucine-zi\npper protein family,Null mutant is viable; expression of eno\nlase genes is reduced three-fivefold in null mutant; gcr1 ty\ne7 double deletion mutants exhibit additive defects in enola\nse expression. TYE7 was isolated as a dominant suppressor of\n gcr1 mutations\n mSIT1:Siderophore Iron Transport,ferrioxamine B permease,Viable. C\nells deleted from the gene are unable to take up ferrioxamin\ne B\n Cond130:rml2(**13)\n mATX1:antioxidant protein and metal homeostasis factor, protects a\ngainst oxygen toxicity,copper binding homeostasis protein (p\nutative),hypersensitive toward paraquat (a generator of supe\nroxide anion)\n Cond121:qcr2(haploid)\n mFET3:FET3 encodes a ferro-O2-oxidoreductase that is part of the h\nigh-affinity iron transport system,multicopper oxidase,The n\null mutant is viable but defective for high affinity Fe(II) \nuptake. The null mutant is inviable when environmental iron \nis limiting.\n Cond89:isw2\n mPDR3:Zinc-finger transcription factor related to Pdr1p,,pleiotrop\nic drug resistance\n Cond4:afg3(haploid)\n mFRE1:Ferric (and cupric) reductase,cupric reductase , ferric redu\nctase,Null mutant is viable, fre1-1 mutants are deficient in\n the uptake of ferric iron and are extremely sensitive to ir\non deprivation\n mYBL049W:Unknown ,, Unknown\n mFTR1:Plasma membrane iron permease,iron permease,Lacks high affin\nity iron uptake\n mFRE2:Ferric reductase, similar to Fre1p,ferric reductase,\n mFRE3:similar to FRE2,,\n mSNQ2:ABC transporter,ABC transporter,null mutant is viable; overe\nxpression confers multi-drug resistance\n mFRE5:similar to FRE2,,\n mFRE6:similar to FRE2,,\n mYDL222C:Unknown ,, Unknown\n mYOR135C:Unknown ,, Unknown\n mYDR112W:Unknown ,, Unknown\n mVID24:also involved in vacuolar protein targeting,peripheral vesic\nle membrane protein,Null mutant is viable, defective in fruc\ntose-1,6-bisphosphatase dergadation\n mYGR035C:Unknown ,, Unknown\n mSPL2:Suppressor of plc1-delta. Isolated as a dosage suppressor of\n the temperature-sensitive phenotype of a plc1 null mutant. \nAlso suppresses the hyperosmotic-sensitive phenotype of the \nplc1 null mutant.,,Null mutant is viable and shows no obviou\ns phenotype; spl2-delta plc1-delta double mutant fails to gr\now on SCD complete media, but grows on YPD at 25 degrees C\n mYKR049C:Unknown ,, Unknown\n mCCC2:copper-transporting P-type ATPase with similarity to human M\nenkes and Wilsons genes,,Null mutant is viable, exhibits def\nects in respiration and iron uptake\n Cond229:yhr011w(**14)\n Cond113:pet111\n mYMR181C:Unknown ,, Unknown\n mRSB1:Unknown ,, Unknown\n mYGL069C:Unknown ,, Unknown\n mENT4:epsin N-terminal homology-containing protein,,unknown\n mSNZ1:Snooze: stationary phase-induced gene family; may be involve\nd in cellular response to nutrient limitation and growth arr\nest,highly conserved 35 kDa protein that shows increased exp\nression after entry into stationary phase,Null mutant is via\nble, sensitive to 6-azauracil and methylene blue.\n mYMR102C:Unknown ,, Unknown\n mODC2:Hypothetical ORF,mitochondrial 2-oxodicarboxylate transport \nprotein,\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mTIS11:Zinc finger containing homolog of mammalian TIS11, glucose r\nepressible gene,,Null mutant is viable but alters metabolism\n that is reflected by a pH change on YPD plates.\n Cond24:cem1\n mYPL261C:Unknown ,, Unknown\n Cond37:cyt1\n Cond20:bub3(haploid**2)\n mRME1:mediates cell type control of sporulation; negatively regula\ntes IME1 and sporulation,negative regulator of meiosis; dire\nctly repressed by a1-a2 regulator , zinc finger protein,Null\n mutant is viable, rme1 allows alpha/alpha and a/a diploids \nto sporulate, and a and alpha haploids to form viable spores\n in the presence of spo13\n mGPG1:Unknown ,, Unknown\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond91:kim4\n mYTP1:Yeast putative Transmembrane Protein,,Null mutant is viable\n mYOR325W:Unknown ,, Unknown\n mYAL061W:Unknown ,, Unknown\n Cond129:rip1\n Cond103:msu1\n Cond265:ymr293c\n mDSE1:Hypothetical ORF,,\n mHMG1:3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase is\nozyme,3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reduct\nase isozyme,Null mutant is viable, sensitive to compactin, a\n competitive inhibitor of HMG-CoA reductase; hmg1 hmg2 doubl\ne deletion mutants are inviable\n mARN1:Product of gene unknown,,\n mARN2:Siderophore transporter for triacetylfusarinine C,triacetylf\nusarinine C transporter,YHL047c disrupted cells are unable t\no take up and utilize iron from triacetylfusarinine C und fu\nsigen\n mYGL157W:Unknown ,, Unknown\n mYOR289W:Unknown ,, Unknown\n mGPH1:Releases glucose-1-phosphate from glycogen,glycogen phosphor\nylase,Null mutant is viable; haploid cells contain higher le\nvels of intracellular glycogen\n mYAL004W:Unknown ,, Unknown\n mSCW11:Soluble Cell Wall protein,soluble cell wall protein,Null mut\nant is viable but exhibits defects in separation after divis\nion and displays flocculant growth.\n mYPL222W:Unknown ,, Unknown\n mCCC2 mATX1
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Computational Genomics Lab, Tel-Aviv uniresity