Module number 700




Database revision : gnsdb28.10
Date : Tue Feb 25 17:43:46 2003
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Cond226:yhl029c\n Cond287:2-deoxy-D-glucose\n Cond423:YPD_stationary_phase_4_h_ypd-1\n mCPT1:Phospholipid biosynthesis,sn-1,2-diacylglycerol cholinephosp\nhotransferase,Null mutant is viable, cpt1 ept1 double deleti\non mutants are viable\n Jelinski.Jelinski:Regulatory networks revealed by transcriptional profiling of\n damaged Saccharomyces cerevisiae cells: Rpn4 links base exc\nision repair with proteasomes. Mol Cell Biol. 2000 Nov;20(2\n1):8157-67.\n Cond424:YPD_stationary_phase_8_h_ypd-1\n mYGR026W:Unknown ,, Unknown\n Cond901:(11i2)_HBY4_YPGL_NormInt\n Cond895:G2MMS\n Cond47:ecm18(**7)\n RapamycinHap.hap_ram:Rapamycin-modulated transcription defines the subset of nutr\nient-sensitive signaling pathways directly controlled by the\n Tor proteins. Proc Natl Acad Sci U S A. 1999 Dec 21;96(26)\n:14866-70.\n mEPT1:sn-1,2-diacylglycerol ethanolamine- and cholinephosphotranfe\nrase,sn-1,2-diacylglycerol ethanolamine- and cholinephosphot\nranferase,Null mutant is viable\n Cond407:diauxic_shift_timecourse11.5_\n Cond887:t-BuOOH\n Cond29:clb2\n mDFG10:Protein required for filamentous growth, cell polarity, and \ncellular elongation,,Null mutant is viable and defective in \nfilamentous growth\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond13:ase1(**12)\n Cond422:YPD_stationary_phase_2_h_ypd-1\n GCR1.gcr1:Understanding the growth phenotype of the yeast gcr1 mutant \nin terms of global genomic expression patterns. J Bacteriol\n. 2000 Sep;182(17):4970-8.\n mERV14:ER-derived vesicles,14 kDa protein found on ER-derived vesic\nles,Null mutant is viable but exhibits defects in sporulatio\nn (diploids) and bud site selection (haploids). Null mutants\n also retain the bud site selection marker, Axl2p, in the ER\n and exhibit slow recovery from selective to rich media.\n Cond886:g-ray\n mCLB6:role in DNA replication during S phase,B-type cyclin,Null mu\ntant is viable\n Cond175:swi4\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mTUF1:Translation elongation factor Tu, mitochondrial,translation \nelongation factor Tu, mitochondrial,Null mutant is viable, b\nlocks mitochondrial translation and destabilizes mitochondri\nal genome.\n Cond883:5\n Cond284:PMA1(tetpromoter)\n mVRG4:May regulate Golgi function and glycosylation in Golgi,,Null\n mutant is inviable; mutants exist with defects in Golgi-spe\ncific glycosylation, resistance to vanadate, sensitivity to \naminoglycosides, and sporulation defects\n Cond244:ymr010w\n Cond153:sgs1\n Stress.YPDStat:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n Cond283:KAR2(tetpromoter)\n mHNM1:choline transport protein; may also control uptake of nitrog\nen mustard,transporter (permease) for choline and nitrogen m\nustard; share homology with UGA4,Null mutant is viable, but \nhyper-resistant to nitrogen mustard; ctr1,cho1 double null i\ns inviable\n Cond53:erg2\n Cond286:YEF3(tetpromoter)\n Cond:\n mGAS3:Unknown ,, Unknown\n Cond889:4NQO_2\n Cond55:erg4(haploid**7)\n Cond295:Lovastatin\n mYGL160W:Unknown ,, Unknown\n Cond888:MNNG_2\n Cond299:Tunicamycin\n Cond570:cdc15_30\n Cell Cycle.cdc15:Comprehensive identification of cell cycle-regulated genes o\nf the yeast Saccharomyces cerevisiae by microarray hybridiza\ntion. Mol Biol Cell. 1998 Dec;9(12):3273-97.\n COMP.TE:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n COMP.CH:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mRCE1:Protease involved in ras and a-factor terminal proteolysis,p\nrotease,Null mutant is viable, has defects in Ras localizati\non and signaling, and suppresses the activated phenotype of \nthe RAS2val19 allele\n mTOM22:Translocase of Outer Mitochondrial membrane,mitochondrial im\nport receptor protein,Null mutant is inviable\n mERV29:ER Vesicle protein of 29 kDa (apparent MW),ER-Golgi transpor\nt vesicle protein,Null mutant is viable.\n Cond852:30_min\n Stress.dshift:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n mCHO1:phosphatidylserine synthase,phosphatidylserine synthase,The \nnull mutant is viable but grows slowly on minimal medium. Th\ne growth rate of the null mutant on minimal medium can be in\ncreased by supplementing the medium with choline or other ph\nospholipid precursors.\n
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Computational Genomics Lab, Tel-Aviv uniresity