Module number 560




Database revision : gnsdb28.10
Date : Tue Feb 25 17:45:08 2003
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Cond498:wtħ500nMaF,30minlog10(intensity)\n mYPR098C:Unknown ,, Unknown\n mSLX4:Hypothetical ORF,,\n mCOX12:essential during assembly for full cytochrome c oxidase acti\nvity,cytochrome c oxidase subunit VIb,Null mutant is viable,\n grows poorly at room temperature, fails to grow on glycerol\n/ethanol media at 37 degrees\n mSET3:,,\n Cond529:bni1D/wtlog10(intensity)\n Cond494:wtħ5nMaF,30minlog10(intensity)\n mTOS6:Hypothetical ORF,,\n mYDL068W:Unknown ,, Unknown\n mSAM3:S-adenosylMethionine Permease,high affinity S-adenosylmethio\nnine permease,Null mutant is viable but has inability to use\n S-adenosylmethionine as a sulfur source\n mPMT1:Transfers mannose residues from dolichyl phosphate-D-mannose\n to specific serine/threonine residues of proteins in the se\ncretory pathway; acts in complex with Pmt2p,dolichyl phospha\nte-D-mannose:protein O-D-mannosyltransferase,Null mutant is \nviable but shows decrease by 40-50% of in vivo protein O-man\nnosylation; pmt1 pmt2 double mutant shows severe growth defe\nct but residual O-mannosylation activity; the pmt1 pmt2 pmt3\n pmt4 quadruple mutant is inviable\n Cond502:wtħ50nMaF,45minlog10(intensity)\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mYAL066W:Unknown ,, Unknown\n mBUD6:actin interacting rotein,,Null mutant is viable; mutants exh\nibit severe disruption of the actin cytoskeleton; deletion s\ntrains have a depolarized cytoskeleton, mitotic delay, and p\nrobable cytokinesis defects\n mYJL120W:Unknown ,, Unknown\n mBUD7:bud site selection,,Diploid-specific heterogenous bud site s\nelection\n mYOL157C:Unknown ,, Unknown\n mYJL182C:Unknown ,, Unknown\n mYOL015W:Unknown ,, Unknown\n mYKR073C:Unknown ,, Unknown\n Cond146:rvs161(haploid)\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond172:ste4(haploid)\n mYLR236C:Unknown ,, Unknown\n Cond501:wt+/-50nMaF,30minlog10(intensity)\n mUBP5:Putative Ubiquitin-specific protease,ubiquitin-specific prot\nease (putative),Null mutant is viable\n mCKI1:choline kinase,choline kinase,Null mutant is viable\n mYBR032W:Unknown ,, Unknown\n mYLR230W:Unknown ,, Unknown\n mYIL174W:Unknown ,, Unknown\n mCUP1-2:copper-binding metallothionein,copper binding metallothionei\nn,Copper resistance\n Cond60:far1(haploid)\n Cond492:wt+/-0.5nMalphaF,30minlog10(intensity)\n mCRS5:Metallothionein-like protein,metallothionein-like protein,Nu\nll mutant is viable, exhibits increased sensitivity to coppe\nr toxicity\n Mating.Mating:Signaling and circuitry of multiple MAPK pathways revealed b\ny a matrix of global gene expression profiles. Science. 200\n0 Feb 4;287(5454):873-80\n mYDR112W:Unknown ,, Unknown\n mYDR203W:Unknown ,, Unknown\n Cond491:wt+/-0.15nMalphaF,30minlog10(intensity)\n Cond523:ste4D/wtlog10(intensity)\n mTRF4:TRF5 homolog; Involved in mitotic chromsome condensation; as\nsociates with Smc1p and Smc2p,DNA polymerase sigma,Null muta\nnt is viable, cold sensitive, display reduced expression of \nGAL1 and cell cycle box UAS::LacZ fusions, and is inviable i\nn combination with a trfI (hpr1) null mutant. trf4 trf5 doub\nle mutants are inviable. A trf4 (ts) trf5 double mutant is h\nypersensitive to the anti-microtubule agent thiabendazole at\n a semi-permissive temperature. A top1 trf4-ts double mutant\n is defective in the mitotic events of chromosome condensati\non, spindle elongation, and nuclear segregation, but not in \nDNA replication. Overexpression of TRF5 complements the invi\nability of top1 trf4 double mutants.\n mVPS13:vacuolar Protein Sorting,,Null mutant is viable but exhibits\n defects in vacuolar protein sorting.\n mKNS1:protein kinase homolog,protein kinase homolog,viable\n mRDS3:Unknown ,, Unknown\n mVPS16:vacuolar sorting protein,,Null mutant is viable, has a sever\ne defect in vacuolar protein sorting, is temperature sensiti\nve for growth, displays grossly abnormal vacuolar morphology\n, and possesses a defect in alpha-factor processing\n mYNL146W:Unknown ,, Unknown\n mPRP16:putative ATP-binding protein,ATP-binding protein (putative),\nNull mutant is inviable\n mYNL043C:Unknown ,, Unknown\n mPFK26:6-Phosphofructose-2-kinase,6-phosphofructose-2-kinase,Null m\nutant is viable; on pyrvuate medium, no fructose 2,6-P2 is d\netectable in mutant\n mPTR2:Functions in transport of small peptides into the cell,pepti\nde transporter,Null mutant is viable\n
this is an automaticly generated SAMBA report
Computational Genomics Lab, Tel-Aviv uniresity