Module number 334




Database revision : gnsdb28.10
Date : Tue Feb 25 17:30:32 2003
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mYGR273C:Unknown ,, Unknown\n mYAR061W:Unknown ,, Unknown\n mAPI2:Unknown ,, Unknown\n mYPL251W:Unknown ,, Unknown\n mYDL068W:Unknown ,, Unknown\n mYAR029W:Unknown ,, Unknown\n mSDL1:L-serine dehydratase,L-serine dehydratase,\n mYLR091W:Unknown ,, Unknown\n GCR1.gcr1:Understanding the growth phenotype of the yeast gcr1 mutant \nin terms of global genomic expression patterns. J Bacteriol\n. 2000 Sep;182(17):4970-8.\n mYAL066W:Unknown ,, Unknown\n Cond670:DES459_(mec1)_-_mock_irradiation_-_5_min\n mYMR122C:Unknown ,, Unknown\n mMPH3:Unknown ,, Unknown\n mMAL12:Part of the complex locus MAL1; functional in S288C,maltase,\nDefective maltose fermentation\n mYGL217C:Unknown ,, Unknown\n mABM1:aberrant microtubules,,\n mYKL223W:Unknown ,, Unknown\n mAST1:Protein involved in targeting of plasma membrane [H+]ATPase,\n,multicopy AST1 suppresses pma1 alleles defective for target\ning\n mYGR212W:Unknown ,, Unknown\n Cond:\n mYPL136W:Unknown ,, Unknown\n COMP.:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mYLR122C:Unknown ,, Unknown\n mYBR300C:Unknown ,, Unknown\n mROM2:Gdp-GTP Exchange Protein (GEP) for the Rho1p Small GTP-bindi\nng Protein; high copy suppressor of cik1 and kar3 deletion m\nutants.,,Null mutant is viable but shows temperature- and co\nld-sensitive growth defects at 37 and 11 degrees, increased \nsensitivity to benomyl, and elongated buds and abnormal mati\nng projections at the permissive temperature; synthetically \nlethal with rom1\n mYHL018W:Unknown ,, Unknown\n Cond268:yor009w\n Cond900:(11i1)_S150-2B_YPGL_NormInt\n mEST1:Telomere elongation protein (ever shorter telomeres),,Null m\nutant is viable, exhibits a senescence phenotype due to the \ngradual loss of sequences essential for telomere function le\nading to a progressive decrease in chromosomal stability and\n subsequent cell death\n Cond111:pcl6\n mRPS26A:Homology to rat S26,ribosomal protein S26A,Null mutant is vi\nable and grows slowly\n TorRama.Series0:Partitioning the transcriptional program induced by rapamyci\nn among the effectors of the Tor proteins. Curr Biol. 2000 D\nec 14-28;10(24):1574-81\n mYLR445W:Unknown ,, Unknown\n mCAT8:Zinc-cluster protein involved in activating gluconeogenic ge\nnes; related to Gal4p,zinc-cluster protein involved in activ\nating gluconeogenic genes; related to Gal4p,Null mutant is v\niable but unable to grow on non-fermentable carbon sources d\nue to failure to derepress all major gluconeogenic enzymes; \noverexpression of Cat8p suppress inability of snf1 and snf4 \nmutants to grow on ethanol\n mYMR306C-A:Unknown ,, Unknown\n mYJR037W:Unknown ,, Unknown\n mYDR065W:Unknown ,, Unknown\n mYNR062C:Unknown ,, Unknown\n mYJR128W:Unknown ,, Unknown\n mYLR140W:Unknown ,, Unknown\n mCDC55:Involved in cellular morphogenesis,protein phosphatase 2A re\ngulatory subunit B,abnormally elongated buds, delay or parti\nal block of septation and/or cell separation; deletion mutan\nt is cold-sensitive\n mYOR379C:Unknown ,, Unknown\n Cond871:yhe710-ss\n mDNA43:Protein required for S-phase (DNA synthesis) initiation or c\nompletion,,Null mutant is inviable; conditional allele demon\nstrates cell-cycle arrest at the restrictive temperature wit\nh a large-budded terminal phenotype\n mSOH1:Soh1p has limited sequence similarity to RNA polymerases and\n interacts with a DNA repair protein, Rad5p, in a two-hybrid\n system assay; may provide a link between recombination in d\nirect repeats and transcription,,Null mutant is viable, show\ns 10-fold increase in recombination. soh1 mutants were origi\nnally identified as suppressors of hyperrecombination hpr1 m\nutants. Soh1p may functionally interact with components of t\nhe RNA polymerase II complex as suggested from the synthetic\n lethality observed in soh1 rpb delta 104, soh1 rpb2, and so\nh1 sua7 double mutants.\n mYJR038C:Unknown ,, Unknown\n mYAR060C:Unknown ,, Unknown\n mYLR123C:Unknown ,, Unknown\n mYKL036C:Unknown ,, Unknown\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mUFD4:Ubiquitin Fusion Degradation,,Null is viable; defective in p\nroteolysis of fusion proteins containing a 'nonremovable' N-\nterminal ubiquitin moiety\n mYHL005C:Unknown ,, Unknown\n mYER085C:Unknown ,, Unknown\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mYDL187C:Unknown ,, Unknown\n mKCC4:involved in septin organization,S. pombe Nim1 homolog , prot\nein kinase,Null mutant is viable\n mYJL028W:Unknown ,, Unknown\n mYMR324C:Unknown ,, Unknown\n mVPS61:Unknown ,, Unknown\n mYDR360W:Unknown ,, Unknown\n mYOR325W:Unknown ,, Unknown\n mYGL182C:Unknown ,, Unknown\n mIRE1:Involved in myo-inositol biosynthesis; implicated as the sen\nsor of unfolded proteins in the ER that initiates transmitta\nnce of the unfolded protein signal from the ER to the nucleu\ns,endoribonuclease , serine-threonine kinase , transmembrane\n protein,Null mutant is viable, myo-inositol auxotroph; IRE1\n is essential for viability under stress conditions that cau\nse unfolded proteins to accumulate in the ER\n mYLR232W:Unknown ,, Unknown\n mYLL030C:Unknown ,, Unknown\n Cond84:hst3\n mYGL034C:Unknown ,, Unknown\n mIME1:Transcriptional activator of meiotic gene expression.,,The n\null mutant is viable. Diploids homozygous for the null mutat\nion lack premeiotic DNA synthesis and do not sporulate; thes\ne phenotypes are recessive.\n mYBL073W:Unknown ,, Unknown\n mYAR047C:Unknown ,, Unknown\n mYMR317W:Unknown ,, Unknown\n mMEI5:Meiotic protein required for synapsis and meiotic recombinat\nion,,\n mYCL063W:Unknown ,, Unknown\n mNKP2:Unknown ,, Unknown\n mYGL118C:Unknown ,, Unknown\n mYOR268C:Unknown ,, Unknown\n mYDR332W:Unknown ,, Unknown\n mYIL025C:Unknown ,, Unknown\n mYOR050C:Unknown ,, Unknown\n mYCR001W:Unknown ,, Unknown\n
this is an automaticly generated SAMBA report
Computational Genomics Lab, Tel-Aviv uniresity