Module number 2856




Database revision : gnsdb28.10
Date : Tue Feb 25 17:35:12 2003
How to read this figure?


mSEC4:Involved in transport and fusion of post-Golgi secretory ves\nicles to the plasma membrane,ras homolog , small GTP binding\n protein,null is inviable; conditional mutants show defects \nin secretion and accumulation of post-Golgi vesicles under n\non-permissive conditions\n Cond899:RPN4_MMS__\n Cond277:AUR1(tetpromoter)\n PEX.Pex:Transcriptome profiling to identify genes involved in peroxi\nsome assembly and function. J Cell Biol. 2002 Jul 22;158(2)\n:259-71.\n mVPS55:Unknown ,, Unknown\n UME6.ume6:The Ume6 regulon coordinates metabolic and meiotic gene expr\nession in yeast. Proc Natl Acad Sci U S A. 2002 Oct 15;99(2\n1):13431-6.\n Jelinski.Jelinski:Regulatory networks revealed by transcriptional profiling of\n damaged Saccharomyces cerevisiae cells: Rpn4 links base exc\nision repair with proteasomes. Mol Cell Biol. 2000 Nov;20(2\n1):8157-67.\n mPLB1:Responsible for the production of the deacylation products o\nf phosphatidylcholine and phosphatidylethanolamine but not p\nhosphatidylinositol,phospholipase B (lypophospholipase),Null\n mutant is viable but releases greatly reduced levels of pho\nsphatidylcholine and phosphatidylethanolamine metabolites\n tcl1.tcl1:The genome-wide expression response to telomerase deletion i\nn Saccharomyces cerevisiae. Proc Natl Acad Sci U S A. 2002 \nJul 9;99(14):9316-21.\n Cond248:ymr030w\n mPMR1:Ca++-Pump, ATPase,Ca2+ ATPase,pmr1 null mutants suppress ypt\n1-1\n mDIA1:may be involved in invasive growth, pseudohyphal growth,,Nul\nl mutant is viable and causes invasive growth in haploids an\nd pseudohyphal growth in diploids\n Meiosis.Series0:The core meiotic transcriptome in budding yeasts. Nat Genet\n. 2000 Dec;26(4):415-23.\n Cond887:t-BuOOH\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mYDR319C:Unknown ,, Unknown\n GCR1.gcr1:Understanding the growth phenotype of the yeast gcr1 mutant \nin terms of global genomic expression patterns. J Bacteriol\n. 2000 Sep;182(17):4970-8.\n mMSC3:Meiotic Sister-Chromatid recombination,,\n mYJL017W:Unknown ,, Unknown\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond874:30min\n Cond883:5\n Cond833:tlc1_Expt.2_Passage_5\n Cond898:RPN4\n mTFP3:vacuolar ATPase V0 domain subunit c' (17 kDa),vacuolar ATPas\ne V0 domain subunit c' (17 kDa) , vacuolar H(+) ATPase 17 kD\na subunit C , vacuolar ATPase V0 domain subunit c' (17 kDa) \n, vacuolar H(+) ATPase 17 kDa subunit C,Null mutant is viabl\ne, defective in vacuolar acidification, high copy TFP3 confe\nrs resistance to trifluoperazine\n Cond940:6h\n Calcin.Ca/Ca:Genome-wide analysis of gene expression regulated by the cal\ncineurin/Crz1p signaling pathway in Saccharomyces cerevisiae\n. J Biol Chem. 2002 Aug 23;277(34):31079-88\n Cond947:W303_YPA\n Cond873:10min\n Cond805:Ca/Ca+FK30'\n mSUR1:Involved in maintenance of phospholipid levels,integral memb\nrane protein , similar to YBR161w, Hoc1p, and Och1p , integr\nal membrane protein , similar to YBR161w, Hoc1p, and Och1p ,\n integral membrane protein , similar to YBR161w, Hoc1p, and \nOch1p,Null mutant is viable, calcium sensitive at 37 degrees\n C on YPD but calcium tolerant at 26 degrees C, accumulates \ngreatly reduced levels of several mannosylated sphingolipids\n; sur1 mutations have been isolated based on their ability t\no suppress certain phenotype of rvs161 mutants including red\nuced viability upon starvation and sensitivies to unrelated \ndrugs; SUR1 is a high copy suppressor of the calcium sensiti\nvity of csg2 mutants\n Cond937:t=0\n Cond888:MNNG_2\n mYOR385W:Unknown ,, Unknown\n Cond903:(77i2)_S150-2B_YPGL_NormInt\n Cond934:8h\n mYOR086C:Unknown ,, Unknown\n COMP.TE:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mPRP12:Integral membrane mitochondrial protein,integral membrane pr\notein,Null mutant is viable but shows increased rate of DNA \nescape from mitochondria to the nucleus and, in some strains\n, shows a growth defect on nonfermentable carbon sources; rn\na12-1 is a dominant, thermosensitive allele that results in \ndefects in RNA maturation at the restrictive temperature; ym\ne1 cold sensitivity is suppressed by prp1; yme1 prp12 double\n mutant has synthetic growth defect on ethanol-glycerol medi\num at 30 degrees\n mYLR454W:Unknown ,, Unknown\n mYGR146C:Unknown ,, Unknown\n Cond839:expt2\n mYIR044C:Unknown ,, Unknown\n Cond939:4h\n
this is an automaticly generated SAMBA report
Computational Genomics Lab, Tel-Aviv uniresity