Module number 2786




Database revision : gnsdb28.10
Date : Tue Feb 25 17:35:19 2003
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Jelinski.Jelinski:Regulatory networks revealed by transcriptional profiling of\n damaged Saccharomyces cerevisiae cells: Rpn4 links base exc\nision repair with proteasomes. Mol Cell Biol. 2000 Nov;20(2\n1):8157-67.\n Cond717:t2-SSD1\n Cond901:(11i2)_HBY4_YPGL_NormInt\n mTRR1:Thioredoxin reductase,thioredoxin reductase,Null mutant is v\niable but grow slowly; trr1 mutations are sensitive to hydro\ngen peroxide and activate Mlu1 cell cycle box (MCB)- and Swi\n4/Swi6 cell cycle box (SCB)-dependent reporter genes in swi6\n null mutants.\n Cond896:STAT\n Cond716:t2+SSD1wt\n mORM1:Product of gene unknown,,\n mGDS1:involved in nuclear control of mitochondria,,Null mutant is \nviable, shows partial impairment of growth on medium contain\ning glycerol as the carbon source. Overexpxression suppresse\ns NAM9-1 glycerol deficient phenotype\n Cond708:t0+SSD1\n Cond418:YPD_1_d_ypd-2\n mPMT1:Transfers mannose residues from dolichyl phosphate-D-mannose\n to specific serine/threonine residues of proteins in the se\ncretory pathway; acts in complex with Pmt2p,dolichyl phospha\nte-D-mannose:protein O-D-mannosyltransferase,Null mutant is \nviable but shows decrease by 40-50% of in vivo protein O-man\nnosylation; pmt1 pmt2 double mutant shows severe growth defe\nct but residual O-mannosylation activity; the pmt1 pmt2 pmt3\n pmt4 quadruple mutant is inviable\n Cond725:t4-SSD1,M31\n mYKL084W:Unknown ,, Unknown\n GCR1.gcr1:Understanding the growth phenotype of the yeast gcr1 mutant \nin terms of global genomic expression patterns. J Bacteriol\n. 2000 Sep;182(17):4970-8.\n mYHR181W:Unknown ,, Unknown\n mLAT1:Dihydrolipoamide acetyltransferase component (E2) of pyruvat\ne dehydrogenase complex,pyruvate dehydrogenase complex dihyd\nrolipoamide acetyltransferase component (E2),Null mutant is \nviable\n Stress.YPD:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n Cond712:t4+SSD1\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond874:30min\n Cond723:t2-SSD1,M31\n mGSF2:Glucose Signaling Factor,,A Tn3 insertion into this gene cau\nses hypersensitivity to the cell surface polymer perturbing \nagent calcofluor white; Defective in glucose repression; mut\nants decrease transcriptional repression by MIG1; alter gluc\nose-regulated subunit interactions within the Snf1 protein k\ninase complex; the effects of eff1 and eff2 on SUC2 repressi\non are strongly synergistic.\n mYDR531W:Unknown ,, Unknown\n Cond719:t4-SSD1\n mITR2:member of sugar transporter superfamily,myo-inositol transpo\nrter,Null mutant is viable\n mVMA5:42 kDa subunit of V1 sector,V1 sector hydrophilic subunit C \n, vacuolar ATPase V1 domain subunit C (42 kDa) , vacuolar H-\nATPase , V1 sector hydrophilic subunit C , vacuolar ATPase V\n1 domain subunit C (42 kDa) , vacuolar H-ATPase,Null mutant \nis viable; certain vma5 mutations show allele-specific synth\netic lethality with cdc24-ls mutants\n Cond724:t4+SSD1,H44\n Cond714:t0+SSD1wt\n mRRP40:Ribosomal RNA Processing,,The null mutant is inviable and de\nfective in 3' processing of 5.8S rRNA\n Cond889:4NQO_2\n mGCS1:Zn-finger-containing protein that functions as ADP-ribosylat\nion factor GTPase-activating protein and is involved in regu\nlating vesicle transport,ADP-ribosylation factor GTPase-acti\nvating protein (ARF GAP),Null mutant is cold-sensitive for r\neentry into mitotic cell cycle from stationary phase and sho\nws inefficient secretion of invertase\n COMP.:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mADO1:adenosine kinase,adenosine kinase,\n mTEF4:Translation elongation factor EF-1gamma,translation elongati\non factor EF-1gamma,\n mPHO13:p-nitrophenyl phosphatase,p-nitrophenyl phosphatase,Null mut\nant is viable\n mSWP1:oligosaccharyl transferase glycoprotein complex, delta subun\nit,oligosaccharyl transferase glycoprotein complex, delta su\nbunit,lethal\n mHXT2:hexose transporter,high affinity hexose transporter-2,Null m\nutant is viable\n Cond718:t4+SSD1wt\n Cond897:STATMMS\n Cond888:MNNG_2\n mKRE9:cell wall beta-glucan assembly,,Null mutant is viable, assoc\niated with growth defects, altered cell wall, aberrant multi\nply budded morphology, mating defects; exhibits double mutan\nt lethality in combination with knh1, kre1, kre6, or kre11 m\nutants; killer toxin resistant; reduction in cell wall (1---\n-6)-beta-glucan\n Cond900:(11i1)_S150-2B_YPGL_NormInt\n Cell Cycle.cdc15:Comprehensive identification of cell cycle-regulated genes o\nf the yeast Saccharomyces cerevisiae by microarray hybridiza\ntion. Mol Biol Cell. 1998 Dec;9(12):3273-97.\n Cond588:cdc15_230\n Cond709:t0+Vec\n mYMR010W:Unknown ,, Unknown\n mLCB2:Involved in sphingolipid biosynthesis; may catalyze the firs\nt step in biosynthesis of long-chain sphingolipids,serine pa\nlmitoyltransferase component (putative),Auxotrophic for long\n-chain component of sphingolipids; some mutations can suppre\nss the Ca2+-sensitive mutant csg2\n Cond710:t2+SSD1\n mYIL039W:Unknown ,, Unknown\n
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Computational Genomics Lab, Tel-Aviv uniresity