Module number 2654




Database revision : gnsdb28.10
Date : Tue Feb 25 17:27:40 2003
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mPET127:May be component of mitochondrial translation system with ro\nle in promoting accuracy of translational initiation or may \nplay a role in RNA surveillance or RNA processing in mitocho\nndria,mitochondrial membrane protein,Null mutant is viable; \npet127 mutations suppress pet122 mutations and have pleiotro\npic effects on stability and 5'-end processing of mitochondr\nial RNAs; overexpression of PET127 blocks respiration and ca\nuses cells to lose mtDNA\n mYDR442W:Unknown ,, Unknown\n mRPS9A:Homology to rat S9 and E.coli S4,ribosomal protein S9A (S13)\n (rp21) (YS11),\n Cond808:Na15'\n Cond871:yhe710-ss\n mPET309:Involved in expression of mitochondrial COX1 by regulating t\nranslation of COX1 mRNA and by affecting transcription or st\nability of COX1 mRNAs,,petite; unable to grow on non-ferment\nable carbon sources\n Cond446:YAP1_overexpression\n mCCZ1:calcium caffeine zinc sensitivity,,Null mutant is viable, bu\nt is sensitive to caffeine, calcium and zinc; no sporulation\n in homozygous null diploids\n mYJL083W:Unknown ,, Unknown\n Cond916:(99i5)__HBY4_YPGL+G_NormInt\n mMRP1:shows allele-specific genetic interactions with pet122 and p\net123,37 kDa mitochondrial ribosomal protein,defective mitoc\nhondrial protein synthesis; absence of a and b type cytochro\nmes; reduced levels of mitochondrial 15 S rRNA; defective pr\nocessing of apocytochrome b intron; convert to rho- and rho0\n at high frequency\n mLSM8:Like Sm-B protein,snRNP protein,Null mutant is inviable\n mMEC3:Involved in checkpoint control and DNA repair,,Null mutant i\ns viable\n mFIL1:Putative mitochondrial ribosome recycling factor,mitochondri\nal ribosome recycling factor (putative),Null mutant is viabl\ne but grows slowly; growth is further impaired by addition o\nf diepoxybutane or mitomycin C to medium. Null mutant also e\nxhibits decreased abundance of mitochondrial proteins.\n mYML050W:Unknown ,, Unknown\n Stress.Various:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n COMP.:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond812:Na+FK15'\n Calcin.Na:Genome-wide analysis of gene expression regulated by the cal\ncineurin/Crz1p signaling pathway in Saccharomyces cerevisiae\n. J Biol Chem. 2002 Aug 23;277(34):31079-88\n mYCR060W:Unknown ,, Unknown\n mELM1:cell morphology,protein kinase,formation of expanded, branch\ned chains of elongated cells; grow invasively under the surf\nace of agar medium\n GCR1.gcr1:Understanding the growth phenotype of the yeast gcr1 mutant \nin terms of global genomic expression patterns. J Bacteriol\n. 2000 Sep;182(17):4970-8.\n mFYV4:Function required for Yeast Viability on toxin exposure,,Nul\nl phenotype is K1 killer toxin hypersensitive\n Cond903:(77i2)_S150-2B_YPGL_NormInt\n Cond676:DES460_vs._DES460_YPD_log_phase_control\n mYAL045C:Unknown ,, Unknown\n mSAC3:involved in processes affecting the actin cytoskeleton and m\nitosis,leucine permease transcriptional regulator,Null mutan\nt is viable, grows more slowly and is larger than wild-type \ncells; exhibits increased benomyl resistance; in contrast to\n sac3-1, sac3 null mutants do not suppress the temperature a\nnd osmosensitivity of act1-1 mutants\n TorRama.Series0:Partitioning the transcriptional program induced by rapamyci\nn among the effectors of the Tor proteins. Curr Biol. 2000 D\nec 14-28;10(24):1574-81\n Calcin.NaFK:Genome-wide analysis of gene expression regulated by the cal\ncineurin/Crz1p signaling pathway in Saccharomyces cerevisiae\n. J Biol Chem. 2002 Aug 23;277(34):31079-88\n mYNL122C:Unknown ,, Unknown\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond902:(77i1)_HBY4_YPGL_NormInt\n mSWF5:Unknown ,, Unknown\n
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Computational Genomics Lab, Tel-Aviv uniresity