Module number 2352




Database revision : gnsdb28.10
Date : Tue Feb 25 17:14:37 2003
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Cond694:gal10+gal\n Cond430:YPD_stationary_phase_7_d_ypd-1\n PEX.Pex:Transcriptome profiling to identify genes involved in peroxi\nsome assembly and function. J Cell Biol. 2002 Jul 22;158(2)\n:259-71.\n Cond912:(99i1)_S150-2B_YPGL_NormInt\n Cond163:ssn6(haploid)\n Cond871:yhe710-ss\n Cond907:(83i1)_S150-2B_YPGL_NormInt\n Cond844:expt7\n mCTA1:catalase A,catalase A,Null mutant is viable and heat sensiti\nve\n mDCI1:Delta(3,5)-delta(2,4)-dienoyl-CoA isomerase,delta(3,5)-delta\n(2,4)-dienoyl-CoA isomerase,\n mYIL163C:Unknown ,, Unknown\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond841:expt4\n gala.+gal:Integrated genomic and proteomic analyses of a systematicall\ny perturbed metabolic network. Science. 2001 May 4;292(5518\n):929-34.\n GCR1.gcr1:Understanding the growth phenotype of the yeast gcr1 mutant \nin terms of global genomic expression patterns. J Bacteriol\n. 2000 Sep;182(17):4970-8.\n mFDH2:Unknown ,, Unknown\n Cond417:YPD_12_h_ypd-2\n gala.-gal:Integrated genomic and proteomic analyses of a systematicall\ny perturbed metabolic network. Science. 2001 May 4;292(5518\n):929-34.\n Stress.YPD:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mPEX11:May promote peroxisomal proliferation by participating in pe\nroxisomal elongation or fission or segregation of peroxisome\ns to daughter cells,peroxisomal membrane protein,Null mutant\n is viable but shows slower growth on oleate (or perhaps no \ngrowth at all), glycerol and acetate and has one or two very\n large peroxisomes; overexpression of PMP27 produces yeast w\nith an increased number of normal-sized peroxisomes\n Cond415:YPD_8_h_ypd-2\n Cond416:YPD_10_h__ypd-2\n mSPC25:Spindle Pole Component of molecular weight 25kDa,spindle pol\ne component,Null mutant is inviable\n Stress.Carbon:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n Cond687:gal1+gal\n Cond843:expt6\n Stress.YPDStat:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n Cond10:are1,are2(haploid)\n GCN4.gcn4:Transcriptional profiling shows that Gcn4p is a master regul\nator of gene expression during amino acid starvation in yeas\nt. Mol Cell Biol. 2001 Jul;21(13):4347-68.\n mTES1:Thioesterase,acyl-CoA thioesterase,Null mutant is viable\n Cond:\n Cond840:expt3\n mYHR140W:Unknown ,, Unknown\n COMP.:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mYGR243W:Unknown ,, Unknown\n Cond429:YPD_stationary_phase_5_d_ypd-1\n Cond815:Na+FK60'\n Cond677:DES459_(mec1)_-_log_phase_(IR_time_=_0_sample)\n Cond845:expt8\n Cond704:gal80-gal\n mFOX2:peroxisomal multifunctional beta-oxidation protein,multifunc\ntional beta-oxidation protein,mutant lacks 2-enoyl-CoA hydra\ntase and D-3-hydroxyacyl-CoA dehydrogenase activities\n mSNP1:U1snRNP 70K protein homolog,U1snRNP 70K protein homolog,Null\n mutant is inviable in some strain backgrounds and in other \nstrain backgrounds, null mutant is viable, exhibits greatly \nincreased doubling rates, severe temperature sensitivities, \nand defects in nuclear pre-mRNA splicing\n mYNL195C:Unknown ,, Unknown\n Cond705:gal1gal10+gal\n Cond473:WT+/-MMS(DBY747)\n Cond420:YPD_3_d_ypd-2\n TorRama.Series0:Partitioning the transcriptional program induced by rapamyci\nn among the effectors of the Tor proteins. Curr Biol. 2000 D\nec 14-28;10(24):1574-81\n Cond842:expt5\n Calcin.NaFK:Genome-wide analysis of gene expression regulated by the cal\ncineurin/Crz1p signaling pathway in Saccharomyces cerevisiae\n. J Biol Chem. 2002 Aug 23;277(34):31079-88\n mFAA2:acyl-CoA synthetase (long-chain fatty acid CoA ligase) (fatt\ny acid activator 2), activates endogenous but not imported f\natty acids and provides substrates for N-myristoylation,acyl\n-CoA synthetase (fatty acid activator 2),Not essential for v\negetative growth when fatty acid synthase (fas) is active\n Cond689:gal3+gal\n Cond449:glucose_vs._reference_pool_car-1\n mPEX1:member of the AAA-protein family,AAA ATPase,mutant lacks mor\nphologically recognizable peroxisomes and shows mislocalizat\nion of peroxisomal matrix proteins\n Cond452:sucrose_vs._reference_pool_car-1\n
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Computational Genomics Lab, Tel-Aviv uniresity