Module number 2144




Database revision : gnsdb28.10
Date : Tue Feb 25 17:04:13 2003
How to read this figure?


Stress.NitroDepl:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n Cond580:cdc15_150\n Cond575:cdc15_100\n Cond401:Nitrogen_Depletion_1_d\n mMF(ALPHA)2:alpha mating factor,alpha mating factor,Null mutant is viabl\ne.\n mTOS4:Hypothetical ORF,,\n Cond589:cdc15_240\n mHLR1:LRE1 homolog,,Null mutant is viable.\n Cond574:cdc15_90\n Cond583:cdc15_180\n Cond578:cdc15_130\n mAPN2:AP endonuclease 2, homolog of human HAP1 and E. coli exoIII,\nAP endonuclease,The apn2 null mutant is viable and is not MM\nS sensitive. The apn1 apn2 null mutant, however, is extremel\ny sensitive to MMS, shows highly elevated frequencies of MMS\n induced mutagenesis, and is defective in the repair of abas\nic sites.\n Cond581:cdc15_160\n Cond576:cdc15_110\n mSLY41:multicopy suppressor of ypt1 deletion,chloroplast phosphate \ntransporter homolog,Null mutant is viable\n Cond572:cdc15_70\n mALG1:beta-1,4-mannosyltransferase,beta-1,4-mannosyltransferase,Nu\nll mutant is inviable\n Cond570:cdc15_30\n mLIF1:Ligase Interacting Factor 1; physically interacts with DNA l\nigase 4 protein (Lig4p),,Null mutant is viable but is defici\nent in non-homologous double-strand break repair; inefficien\nt in sporulation; LIG4 protein destabilization\n Cond584:cdc15_190\n Cell Cycle.cdc15:Comprehensive identification of cell cycle-regulated genes o\nf the yeast Saccharomyces cerevisiae by microarray hybridiza\ntion. Mol Biol Cell. 1998 Dec;9(12):3273-97.\n mDON1:prospore membrane localizing protein,,\n Cond579:cdc15_140\n mRME1:mediates cell type control of sporulation; negatively regula\ntes IME1 and sporulation,negative regulator of meiosis; dire\nctly repressed by a1-a2 regulator , zinc finger protein,Null\n mutant is viable, rme1 allows alpha/alpha and a/a diploids \nto sporulate, and a and alpha haploids to form viable spores\n in the presence of spo13\n mPIN4:[PSI+] induction,,Other phenotypes: overexpression of PIN4 a\nllows for the induction of the [PSI+] prion by Sup35p overpr\noduction in the strains cured of [PIN+].\n Cond588:cdc15_230\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mBFA1:Byr four alike,,Null mutant is viable; mutants are sensitive\n to microtubule inhibitors, exhibit defects in mitotic check\npoints, and exhibit moderate defects in mating efficiency\n Cond582:cdc15_170\n mCVT9:Oligomeric, coiled-coil, peripheral membrane protein,Oligome\nric, coiled-coil, peripheral membrane protein required for s\ntable binding of precursor API to its target membrane.,cvt9 \nis defective in maturation of the vacuolar protein, aminopep\ntidase I and exhibits minor defects in autophagy , cvt9 is d\nefective in vacuolar delivery of aminopeptidase I and peroxi\nsome degradation but is not needed for macroautophagy. The n\null mutant is viable and is relatively starvation-insensitiv\ne.\n mSPR1:Sporulation regulated genes,exo-1,3-beta-glucanase, sporulat\nion-specific,Fail to hydrolyze p-nitrophenyl-beta-D-glucanas\ne or laminarin; mutant spores exhibit reduced thermoresistan\nce\n Cond586:cdc15_210\n
this is an automaticly generated SAMBA report
Computational Genomics Lab, Tel-Aviv uniresity