Module number 2096




Database revision : gnsdb28.10
Date : Tue Feb 25 17:07:27 2003
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UME6.ume6:The Ume6 regulon coordinates metabolic and meiotic gene expr\nession in yeast. Proc Natl Acad Sci U S A. 2002 Oct 15;99(2\n1):13431-6.\n mLYS5:aminoadipate-semialdehyde dehydrogenase small subunit (alpha\n-aminoadipate reductase),aminoadipate-semialdehyde dehydroge\nnase small subunit (alpha-aminoadipate reductase),Lysine req\nuiring\n Jelinski.Jelinski:Regulatory networks revealed by transcriptional profiling of\n damaged Saccharomyces cerevisiae cells: Rpn4 links base exc\nision repair with proteasomes. Mol Cell Biol. 2000 Nov;20(2\n1):8157-67.\n mTRR2:mitochondrial thioredoxin reductase,thioredoxin reductase,Nu\nll mutant is viable, increased sensitivity to hydrogen perox\nide\n Cond896:STAT\n mDFG10:Protein required for filamentous growth, cell polarity, and \ncellular elongation,,Null mutant is viable and defective in \nfilamentous growth\n Cond882:zero3\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mFUN34:Highly homologous to Ycr010p and similar to Yarrowia lipolyt\nica glyoxylate pathway regulator GPR1 (see MIPS),transmembra\nne protein (putative),Null mutant is viable\n mYGR223C:Unknown ,, Unknown\n mYMR090W:Unknown ,, Unknown\n mSNO3:SNZ3 proximal ORF, stationary phase induced gene family,,Nul\nl mutant is viable.\n Cond946:W303ume6_YPD\n mBNS1:bypasses need for SPO12,,Null mutant is viable and exhibits \nno obvious meiotic defects. When overexpressed, BNS1 can par\ntially suppress the meiotic defect of spo12/spo12 deletion m\nutants.\n mRME1:mediates cell type control of sporulation; negatively regula\ntes IME1 and sporulation,negative regulator of meiosis; dire\nctly repressed by a1-a2 regulator , zinc finger protein,Null\n mutant is viable, rme1 allows alpha/alpha and a/a diploids \nto sporulate, and a and alpha haploids to form viable spores\n in the presence of spo13\n mYHR083W:Unknown ,, Unknown\n mUGX2:Product of gene unknown,,\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mPET54:translational activator of cytochrome c oxidase subunit III;\n required for splicing of cytochrome c oxidase subunit I mRN\nA,translational activator of cytochrome C oxidase subunit II\nI; required for splicing of cytochrome c oxidase subunit I \nmRNA,petite; unable to grow on non-fermentable carbon source\ns\n mAPS2:Related to the sigma subunit of the mammalian plasma membran\ne clathrin-associated protein (AP-2) complex,clathrin associ\nated protein complex small subunit,null mutant is viable; sl\night effect on chc1-ts cell growth\n Cond883:5\n mYMR073C:Unknown ,, Unknown\n Cond125:ras1\n Cond876:zero2\n Cond880:BCNU\n Cond889:4NQO_2\n Cond574:cdc15_90\n mPCP1:Unknown ,, Unknown\n Cond873:10min\n mCRS5:Metallothionein-like protein,metallothionein-like protein,Nu\nll mutant is viable, exhibits increased sensitivity to coppe\nr toxicity\n mYGR024C:Unknown ,, Unknown\n mMPT5:Product of gene unknown,,Null mutant is viable, temperature \nsensitive\n mYOR228C:Unknown ,, Unknown\n mEMI5:Unknown ,, Unknown\n mYIL059C:Unknown ,, Unknown\n Cond888:MNNG_2\n mCUE1:Cue1p assembles with Ubc7p. Cue1p recruits Ubc7p to the cyto\nsolic surface of the endoplasmic reticulum. Assembly with Cu\ne1p is a prerequisite for the function of Ubc7p,Ubc7p bindin\ng and recruitment protein,Null mutant is viable and shows st\nabilization of ER degradation substrates\n Cond894:G2\n mIAH1:isoamyl acetate-hydrolyzing esterase,isoamyl acetate-hydroly\nzing esterase,The null mutant is viable but cannot hydrolyze\n isoamyl acetate.\n mYPR147C:Unknown ,, Unknown\n Cell Cycle.cdc15:Comprehensive identification of cell cycle-regulated genes o\nf the yeast Saccharomyces cerevisiae by microarray hybridiza\ntion. Mol Biol Cell. 1998 Dec;9(12):3273-97.\n mHCH1:high copy Hsp90 supressor,,Null mutant is viable; when overe\nxpressed, HCH1 is an allele-specific suppressor of hsp82 ts \nmutants\n mRNR3:Ribonucleotide reductase (ribonucleoside-diphosphate reducta\nse) large subunit,ribonucleotide reductase, large (R1) subun\nit,Null mutant is viable\n mYIM1:Mitochondrial inner membrane protease, similar to E. coli le\nader peptidase,protease , similar to E. coli leader peptidas\ne,\n Cond893:SMMS\n mELC1:similar to mammalian elongin C, interacts with elongin A,elo\nngin C transcription elongation factor,\n mPEX2:Required for peroxisome biogenesis,CH3HC4 zinc-binding integ\nral peroxisomal membrane protein,Null mutant is viable but l\nacks morphologically recognizable peroxisomes and shows cyto\nsolic mislocalization of peroxisomal matrix proteins\n
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Computational Genomics Lab, Tel-Aviv uniresity