Module number 2030




Database revision : gnsdb28.10
Date : Tue Feb 25 17:43:43 2003
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Cond547:cln3-1\n Cond498:wtħ500nMaF,30minlog10(intensity)\n mTOS2:Hypothetical ORF,,\n mPOL1:Required for mitotic DNA synthesis, premeiotic DNA synthesis\n, recombination, and full sporulation,DNA polymerase I alpha\n subunit p180,Null mutant is inviable. pol1(ts) mutants show\n blocked cell division at 36 degrees C\n mAXL2:involved in polarity establishment/cellular polarization dur\ning budding,,AXL2 can serve as a multicopy suppressor of rho\n3 and is required for the haploid axial budding pattern of S\n. cerevisiae.\n mSWI4:Involved in cell cycle dependent gene expression,transcripti\non factor,Null mutant is viable, deficient in homothallic sw\nitching, and temperature sensitive\n Cond494:wtħ5nMaF,30minlog10(intensity)\n Cond548:cln3-2\n Cond500:wtħ50nMaF,15minlog10(intensity)\n Cond751: Cell Cycle.alpha:Comprehensive identification of cell cycle-regulated genes o\nf the yeast Saccharomyces cerevisiae by microarray hybridiza\ntion. Mol Biol Cell. 1998 Dec;9(12):3273-97.\n Cond504:wtħ50nMaF,90minlog10(intensity)\n Cond579:cdc15_140\n mPSA1:mannose-1-phosphate guanyltransferase, GDP-mannose pyrophosp\nhorylase,GDP-mannose pyrophosphorylase , mannose-1-phosphate\n guanyltransferase , GDP-mannose pyrophosphorylase , mannose\n-1-phosphate guanyltransferase , GDP-mannose pyrophosphoryla\nse , mannose-1-phosphate guanyltransferase,Null mutant is in\nviable; PSA1 is an extragenic suppressor of alg1 mutants; os\nmotically sensitive--lyses upon transfer to hypertonic condi\ntions\n mRFA3:subunit 3 of replication factor-A,replication factor-A subun\nit 3,Null mutant is inviable and arrests as budded and multi\nply budded cells\n Cell Cycle.ko:Comprehensive identification of cell cycle-regulated genes o\nf the yeast Saccharomyces cerevisiae by microarray hybridiza\ntion. Mol Biol Cell. 1998 Dec;9(12):3273-97.\n Cond519:rst1Drst2Dħ50nMaF,30minlog10(intensity)\n mYJR054W:Unknown ,, Unknown\n GCN4.gcn4:Transcriptional profiling shows that Gcn4p is a master regul\nator of gene expression during amino acid starvation in yeas\nt. Mol Cell Biol. 2001 Jul;21(13):4347-68.\n mGAS1:Glycophospholipid-anchored surface protein,cell surface glyc\noprotein 115-120 kDa,Null mutant is slow growing and exhibit\ns cell wall defects.\n Cond53:erg2\n Cond733: Y-Stre.Acid:Remodeling of yeast genome expression in response to environ\nmental changes. Mol Biol Cell. 2001 Feb;12(2):323-37.\n mCDC9:essential for mitosis and meiosis, dispensable for intrageni\nc recombination, but required for haploidization and spores,\nDNA ligase,cell division cycle blocked at 36 degrees, increa\nsed sensitivity to ultraviolet radiation and bleomycin; temp\nerature sensitive\n Cond507:bni1Dħ50nMaF,90minlog10(intensity)\n mGAS3:Unknown ,, Unknown\n Cond509:kss1Dħ50nMaF,30minlog10(intensity)\n Cond562:alpha77\n Cond496:wtħ50nMaF,30minlog10(intensity)\n Mating.Mating:Signaling and circuitry of multiple MAPK pathways revealed b\ny a matrix of global gene expression profiles. Science. 200\n0 Feb 4;287(5454):873-80\n mPMS1:Required for mismatch repair in mitosis and meiosis, low lev\nels of postmeiotic segregation, and high spore viability, di\nspensable for homeologous recombination,mutL homolog , simil\nar to Mlh1p, associates with Mlh1p, possibly forming a heter\nodimer, Pms1p and Msh1p act in concert to bind to a Msh2p-he\nteroduplex complex containing a G-T mismatch,Null mutant is \nviable; postmeiotic segregation increased\n mMRC1:Mediator of the Replication Checkpoint; required for full ac\ntivation of Rad53p in response to replication stress.,,Null:\n sensitive to hydroxyurea; replication checkpoint defective;\n slower DNA replication than wild type; partial loss of sile\nncing at telomeres and HM loci; synthetic lethal with rad9 n\null, rad53-21, and mec1-21.\n Cond480:WT+/-100mM3AT(SetA)(R491)\n Cond568:alpha119\n mPDS5:Precocious Dissociation of Sister chromatids,,\n Cond495:wtħ15.8nMaF,30minlog10(intensity)\n Cond279:ERG11(tetpromoter)\n COMP.CH:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond508:bni1Dħ50nMaF,120minlog10(intensity)\n Cond768:Acid_60'\n mMSH2:Functions with Pms1p and Pms2/Mlh1p in a complex that intera\ncts with Pms3p/Msh6p to repair single-base and insertion-del\netion mispairs, or Msh3p to repair insertion-deletion mispai\nrs.,mutS homolog,Null mutant is viable. Haploid mutants disp\nlay an 85-fold increased rate of spontaneous mutation to can\navanine resistance. Mutants are defective for gene conversio\nn polarity gradients and high spore viability.\n Cond216:yer044c(haploid)\n Cond550:clb2-1\n Cond482:WT+/-100mM3AT(SetC)(KNY164)\n Cond226:yhl029c\n Cond506:bni1Dħ50nMaF,60minlog10(intensity)\n Cond580:cdc15_150\n mYBR071W:Unknown ,, Unknown\n Cond502:wtħ50nMaF,45minlog10(intensity)\n Cond54:erg3(haploid)\n Cond581:cdc15_160\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Stress.Heat2:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n Cond563:alpha84\n Cond558:alpha49\n Sporulation.Series0:The transcriptional program of sporulation in budding yeast.\n Science. 1998 Oct 23;282(5389):699-705.\n Cond734: SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond501:wt+/-50nMaF,30minlog10(intensity)\n Cond250:ymr031w-a\n Cond582:cdc15_170\n Cond314:Heat_Shock_060_minutes__hs-2\n Y-Stre.Alkali:Remodeling of yeast genome expression in response to environ\nmental changes. Mol Biol Cell. 2001 Feb;12(2):323-37.\n Cond554:alpha21\n Cond73:gyp1\n Cond738:90\n Cond571:cdc15_50\n Cond575:cdc15_100\n Cond776:Alkali_100'\n Cond92:kin3\n Cond294:Itraconazole\n Cond965:ndt80_delete_mid_g/r_ratio_\n mDUN1:DNA damage response,protein kinase,Null mutant is viable, de\nfective in DNA damage repair and in DNA damage-resposive ind\nuction of RNR genes, and sensitive to DNA damaging agents\n Cond737: Cond747: mSPT21:involved in trascriptional regulation of Ty1 LTRs,non-specif\nic DNA binding protein,Null mutant is viable, spt21 mutation\ns suppress Ty insertion mutations\n Cond497:wtħ158nMaF,30minlog10(intensity)\n mHSL1:Negative regulator of swe1 kinase (which regulates cdc28),pr\notein kinase (putative) , similar to S. pombe cdr1/nim1,Nul\nl mutant is viable; synthetically lethal with histone H3 mut\nations; G2 delay\n Cond560:alpha63\n Cond576:cdc15_110\n Cond555:alpha28\n mMSB2:putative integral membrane protein,integral membrane protein\n (putative),multicopy suppressor of cdc24 ts mutation\n Cond493:wtħ1.5nMaF,30minlog10(intensity)\n Cond736: Cond503:wtħ50nMaF,60minlog10(intensity)\n Cond511:GAL-STE4,3hrs.gallog10(intensity)\n Cell Cycle.cdc15:Comprehensive identification of cell cycle-regulated genes o\nf the yeast Saccharomyces cerevisiae by microarray hybridiza\ntion. Mol Biol Cell. 1998 Dec;9(12):3273-97.\n COMP.TE:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cond559:alpha56\n mSWE1:protein kinase homolog,protein kinase homolog,Null mutant is\n viable\n Cond750: fkh1,2sf.Series0:Two yeast forkhead genes regulate the cell cycle and pseudoh\nyphal growth. Nature. 2000 Jul 6;406(6791):90-4.\n Cond551:alpha0\n Cond549:clb2-2\n mYOX1:Homeodomain protein that binds leu-tRNA gene,homeobox-domain\n containing protein,Null mutant is viable\n mPMS1 mMSH2
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Computational Genomics Lab, Tel-Aviv uniresity