Module number 1503




Database revision : gnsdb28.10
Date : Tue Feb 25 17:06:17 2003
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mSFB2:binds to Sed5p and Sec23p by distinct domains,zinc finger pr\notein (putative),\n mSEC4:Involved in transport and fusion of post-Golgi secretory ves\nicles to the plasma membrane,ras homolog , small GTP binding\n protein,null is inviable; conditional mutants show defects \nin secretion and accumulation of post-Golgi vesicles under n\non-permissive conditions\n mYLR346C:Unknown ,, Unknown\n UME6.ume6:The Ume6 regulon coordinates metabolic and meiotic gene expr\nession in yeast. Proc Natl Acad Sci U S A. 2002 Oct 15;99(2\n1):13431-6.\n mYLR012C:Unknown ,, Unknown\n mYJR157W:Unknown ,, Unknown\n mSSS1:involved in transfer of secretory precursors through the end\noplasmic reticulum membrane,ER protein , Sec61 trimeric comp\nlex component , Ssh1 trimeric complex component,Null mutant \nis inviable. Depletion of the Sss1 protein rapidly results i\nn accumulation of multiple secretory or membrane proteins de\nvoid of post-translational modifications. SSS1 overexpressio\nn restores translocation in sec61 mutants.\n mSPI1:Stationary Phase Induced; strongly expressed during stationa\nry phase, and trancription is dependent on MSN2/MSN4.,,\n mYLR162W:Unknown ,, Unknown\n mERR2:enolase-related subtelomeric sequence (ERR1 and ERR2 code fo\nr identical proteins),,\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mYMR323W:Unknown ,, Unknown\n mYNL274C:Unknown ,, Unknown\n mFDH2:Unknown ,, Unknown\n mMST1:mitochondrial threonine-tRNA synthetase,,Null mutant is viab\nle\n mYDR455C:Unknown ,, Unknown\n mGPG1:Unknown ,, Unknown\n mCDC21:cell division cycle blocked at 36 degree C,thymidylate synth\nase,defective in continued replication during S phase of the\n cell cycle; temperature-sensitive thymidylate auxotroph\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mYET1:Yeast BAP31 homolog,yeast endoplasmic reticulum 25 kDa trans\nmembrane protein,Null mutant is viable\n mFIT2:Facilitator of iron transport,Cell wall protein involved in \niron transport,impaired siderophore-iron uptake, activation \nof the major iron -dependent transcription factor, AFT1\n mRFA2:Involved in nucleotide excision repair,29% identical to the \nhuman p34 subunit of RF-A , replication factor RF-A subunit \n2,Null mutant is inviable; arrests as budded and multiply bu\ndded cells; rfa2 (ts) cells have a mutator and a hyper-recom\nbination phenotype and are more sensitive to hydroxyurea and\n methyl-methane-sulfonate than wild-type cells\n mFIT3:Facilitator of Iron Transport,Cell wall protein involved in \niron transport,impaired siderophore iron uptake, activation \nof the major iron-dependent transcription factor, AFT1\n mTRK2:membrane protein; low affinity potassium transport,low affin\nity potassium transport , membrane protein,Null mutant is vi\nable, requires added potassium; trk1 trk2 double mutants are\n viable\n mOST3:Catalyzes the transfer of oligosaccharide from dolichol-olig\nosaccharide donor to consensus glycosylation acceptor sites \n(asparagines) in newly synth. proteins - ER lumen; may enhan\nce oligosacch. transfer to subset of acceptor substrates,oli\ngosaccharyl transferase glycoprotein complex 34 kDa gamma su\nbunit,Null mutant is viable but shows underglycosylation of \nsoluble and membrane-bound glycoproteins and contains less o\nligosaccharyltransferase activity in vitro\n mFLO8:Nuclear protein required for diploid filamentous growth, hap\nloid invasive growth and flocculation; note that S288C strai\nns have a mutation in this gene,transcriptional activator of\n FLO1 (putative),Null mutant is viable; wild-type gene is re\nquired for flocculation and for pseudo-hyphal growth\n Cond125:ras1\n mSCJ1:dnaJ homolog,DnaJ homolog,Null mutant is viable but exhibits\n defects in protein sorting and sensitivity to tunicamycin.\n mDAL2:allantoicase,allantoicase,Allantoin degradation deficient\n Cond947:W303_YPA\n mCLN2:role in cell cycle START,G1 cyclin,Null mutant is viable, ex\nhibits G1 arrest; dominant mutation advances the G(sub)1- to\n S- phase transition and impairs ability of cells to arrest \nin G(sub)1 phase in response to external signals\n mYOR285W:Unknown ,, Unknown\n mYFL019C:Unknown ,, Unknown\n mARN1:Product of gene unknown,,\n mYML122C:Unknown ,, Unknown\n mYPR174C:Unknown ,, Unknown\n mNBP1:Nap1p Binding Protein ,,Null mutant is inviable\n Cond59:erp4\n Cond750: fkh1,2sf.Series0:Two yeast forkhead genes regulate the cell cycle and pseudoh\nyphal growth. Nature. 2000 Jul 6;406(6791):90-4.\n
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Computational Genomics Lab, Tel-Aviv uniresity