Module number 1414




Database revision : gnsdb28.10
Date : Tue Feb 25 17:04:09 2003
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Cond87:isw1\n mMND1:needed for Meiotic Nuclear Divisions,,Null mutant is viable;\n arrests after DNA-replication but before nuclear divisions \nafter shift to sporulation medium.\n Cond936:12h\n Cond871:yhe710-ss\n mGAT3:The amino acid sequence of this ORF is very homologous to th\nat of GAT4/YIR013C.,,\n Meiosis.Series0:The core meiotic transcriptome in budding yeasts. Nat Genet\n. 2000 Dec;26(4):415-23.\n mDTR1:dityrosine transporter MFS-MDR,dityrosine transporter MFS-MD\nR,Null mutant is viable; bisformyl dityrosine accumulates in\n cytoplasm of spores; spore wall dityrosine is significantly\n reduced\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Sporulation.Series0:The transcriptional program of sporulation in budding yeast.\n Science. 1998 Oct 23;282(5389):699-705.\n Cond963:t11.5_g/r_ratio\n mCLB1:Involved in mitotic induction,B-type cyclin,Null mutant is v\niable (lethal in combination with clb2 mutation)\n mYOR365C:Unknown ,, Unknown\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mYLR049C:Unknown ,, Unknown\n mSPO13:Spo13 acts as a transcriptional activator in a one-hybrid as\nsay (see Henninger et al (1996) SPO13 and control of meiotic\n chromosome segregation in Saccharomyces cerevisiae),,Null m\nutant is viable, defective for sporulation; loss of function\n results in a single division during meiosis (with some chro\nmosomes segregating reductionally or aberrantly depending on\n strain background), occurring slightly earlier or at the ti\nme of wild type meiosis I, and dyad asci containing two dipl\noid spores. spo13 rescues the meiotic lethality of early Rec\n- mutants and Rec+ haploids. Gain of function causes a CDC28\n-dependent arrest at M-phase in mitosis and a delay in MI nu\ncleardivision.\n mYDR317W:Unknown ,, Unknown\n mSPC29:Nuclear import protein,spindle pole body associated protein,\nNull mutant is inviable\n mYNL019C:Unknown ,, Unknown\n mMRL1:Mannose 6-phosphate Receptor Like,,\n Cond940:6h\n mYPL033C:Unknown ,, Unknown\n mCTS2:chitinase,Sporulation-specific chitinase,Null mutant fails t\no form mature asci, synthesis of spore wall surface layers i\ns affected.\n mCWP1:cell wall protein, involved in O and N glycosylation, accept\nor of B1-6 glucan.,cell wall mannoprotein,Null mutant is via\nble, has increased sensitivities to calcoflour white and con\ngo red\n mYUH1:ubiquitin hydrolase,ubiquitin hydrolase,\n Cond937:t=0\n Cond960:t5_g/r_ratio\n Cond934:8h\n mYDR374C:Unknown ,, Unknown\n Cond938:2h\n Cond964:ndt80_delete_early_g/r_ratio\n TorRama.Series0:Partitioning the transcriptional program induced by rapamyci\nn among the effectors of the Tor proteins. Curr Biol. 2000 D\nec 14-28;10(24):1574-81\n Cond961:t7_g/r_ratio\n Cond962:t9_g/r_ratio\n mAPC5:subunit of the Anaphase Promoting Complex; all known APC sub\nunits co-immunoprecipitate with epitope-tagged Apc5,anaphase\n promoting complex (APC) subunit,Null mutant is inviable at \n25 C\n Cond935:10h\n mSPR1:Sporulation regulated genes,exo-1,3-beta-glucanase, sporulat\nion-specific,Fail to hydrolyze p-nitrophenyl-beta-D-glucanas\ne or laminarin; mutant spores exhibit reduced thermoresistan\nce\n mMSH5:dispensable for DNA repair and meiotic intrachromosomal reci\nprocal recombination, required for full reciprocal recombina\ntion between homologs, and spore viability,mutS homolog,Null\n mutant is viable. Diploids lacking the MSH5 gene display de\ncreased levels of spore viability, increased levels of meios\nis I chromosome nondisjuction, and decreased levels of recip\nrocal exchange between, but not within, homologs. Gene conve\nrsion is not reduced. Msh5 mutants are phenotypically simila\nr to mutants in the meiosis-specific gene MSH4. msh5 is epis\ntatic to msh4, suggesting they act in the same pathway.\n
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Computational Genomics Lab, Tel-Aviv uniresity