Module number 1363




Database revision : gnsdb28.10
Date : Tue Feb 25 17:07:04 2003
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mECM18:ExtraCellular Mutant,,A Tn3 insertion into this gene causes \nhypersensitivity to the cell surface polymer perturbing agen\nt calcofluor white.\n Cond368:dtt_480_min_dtt-2\n mRHR2:DL-glycerol-3-phosphatase,DL-glycerol-3-phosphatase,\n Stress.DTT2:Genomic expression programs in the response of yeast cells t\no environmental changes. Mol Biol Cell. 2000 Dec;11(12):424\n1-57\n mYDR209C:Unknown ,, Unknown\n mYOL037C:Unknown ,, Unknown\n Cond47:ecm18(**7)\n Cond166:ste11(haploid)\n mMET3:ATP sulfurylase,ATP sulfurylase,Null mutant is viable, and i\ns a methionine auxotroph\n Mating.Mating:Signaling and circuitry of multiple MAPK pathways revealed b\ny a matrix of global gene expression profiles. Science. 200\n0 Feb 4;287(5454):873-80\n mBAR1:extracellular protease synthesized in a-cells that cleaves a\nnd inactivates alpha factor,protease , synthesized in a-cell\ns; cleaves and inactivates alpha factor,MATa bar1 cells are \nsupersensitive to the G1 arrest induced by alpha factor\n mMET6:vitamin B12-(cobalamin)-independent isozyme of methionine sy\nnthase (also called N5-methyltetrahydrofolate homocysteine m\nethyltransferase or 5-methyltetrahydropteroyl triglutamate h\nomocysteine methyltransferase),vitamin B12-(cobalamin)-indep\nendent isozyme of methionine synthase (also called N5-methyl\ntetrahydrofolate homocysteine methyltransferase or 5-methylt\netrahydropteroyl triglutamate homocysteine methyltransferase\n),Null mutant is viable, and is a methionine auxotroph\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n Cell Cycle.alpha:Comprehensive identification of cell cycle-regulated genes o\nf the yeast Saccharomyces cerevisiae by microarray hybridiza\ntion. Mol Biol Cell. 1998 Dec;9(12):3273-97.\n mPDR5:multidrug resistance transporter,multidrug resistance transp\norter,pleiotropic drug resistance\n Cond111:pcl6\n mIMG2:required for integrity of mitochondrial genome,,Null mutant \nis viable but shows respiratory deficiency and loss of wild-\ntype mtDNA: conversion to rho- and rho zero petites\n COMP.CH:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mGRE2:induced by osmotic stress; similar to dihydroflavonol 4-redu\nctase from plants,,\n Cond99:med2(haploid)\n mYGR073C:Unknown ,, Unknown\n mSPO11:Dispensable for mitosis, premeiotic DNA synthesis, spindle p\nole body duplication, meiosis I, meiosis II & spores. Requir\ned for chromosome pairing seen by in situ hybridization, dou\nble strand breaks, synaptonemal complexes.,early meiosis-spe\ncific recombination protein,Null mutant is viable, sporulati\non defective; spo11 executes meiosis I early, is rescued by \nspo13 and is epistatic to rad52 spo13; and is classified as \nan early recombination function. It fails to form any recomb\nination intermediates and is partially suppressed by X-irrad\niation (by induced double strand breaks), in meiosis. The sp\no11-1 temperature sensitive mutant is double strand break-, \nrecombination-, and exhibits tripartite synaptonemal complex\n structures at a restrictive temperature, suggesting synapto\nnemal complex formation may be partially independent of doub\nle strand breaks and recombination. mRNA is induced early in\n meiosis; meiosis-specific regulation is dependent on a URS1\n element within the coding region.\n Cond365:dtt_060_min_dtt-2\n Cond124:rad6(haploid)\n Cond19:bub3(**2,8,13)\n Cond289:Cycloheximide\n Cond557:alpha42\n Cond526:ste11D/wtlog10(intensity)\n
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Computational Genomics Lab, Tel-Aviv uniresity