Module number 1295




Database revision : gnsdb28.10
Date : Tue Feb 25 17:07:28 2003
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Cond717:t2-SSD1\n mMCM16:Involved in a nonessential role that governs the kinetochore\n-microtubule mediated process of chromosome segregation,,Nul\nl mutant is viable, exhibits increased sensitivity to the an\nitmitotic drugs benomyl and thiabenzadole; exhibits a high r\nate of chromosome III loss without a significant increase in\n recombination frequency, may exhibit altered kinetochore as\nsembly\n Cond658:DES460_(wt)_-_mock_irradiation_-_5_min\n Cond716:t2+SSD1wt\n mYLL029W:Unknown ,, Unknown\n mDOM34:an ORF of unknown function located in a centromeric region d\nuplicated between chromosomes III and XIV (DOM34 homologue o\nn chromosome III is a pseudogene),,\n mMOG1:Required for nuclear-protein import,nuclear protein that int\neracts with GTP-Gsp1p,Null mutant is viable, temperature sen\nsitive, exhibits defects in nuclear-protein import; MOG1 ove\nrexpression supresses the temperature sensitivity of gsp1 st\nrains; overexpression of NTF2 or GSP1 can suppress the ts ph\nenotype of mog1\n Cond708:t0+SSD1\n Cond725:t4-SSD1,M31\n Cond711:t2+Vec\n mURA4:Third step in pyrimidine biosynthesis pathway,dihydrooratase\n,Null mutant is viable and requires uracil\n mYBR242W:Unknown ,, Unknown\n COMP.KO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mMNN10:Required for mannan synthesis and for polarized growth and b\nud emergence,galactosyltransferase,Null mutant is viable, is\n larger than wild-type cells, is deficient in bud emergence,\n and depends upon an intact morphogenesis checkpoint control\n to survive\n mSFH5:Unknown ,, Unknown\n Cond712:t4+SSD1\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mFOL1:folic acid synthesis,dihydro-6-hydroxymethylpterin pyrophosp\nhokinase , dihydroneopterin aldolase , dihydropteroate synth\netase,essential, induces pseudohyphal growth\n mABZ1:para-aminobenzoate synthase, PABA synthase,para-aminobenzoat\ne synthase (PABA synthase),Null mutant is viable and PABA au\nxotroph\n mYDR119W:Unknown ,, Unknown\n Cond19:bub3(**2,8,13)\n Cond723:t2-SSD1,M31\n mSNF4:involved in release from glucose repression, invertase expre\nssion, and sporulation,associates with Snf1p,Null mutant is \nviable, sucrose nonfermenting; high copy MSI1 and PDE2 parti\nally suppress sporulation defect\n mNUG1:NUclear GTPase,Nuclear GTPase involved in Ribosome biogenesi\ns,Null: dead.\n Cond724:t4+SSD1,H44\n Cond713:t4+Vec\n COMP.:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mFPS1:Suppressor of tps1/fdp1 and member of the MIP family of tran\nsmembrane channels; may be involved in glycerol efflux,glyce\nrol channel protein,Null mutant is viable\n mAAT1:aspartate aminotransferase, mitochondrial,aspartate aminotra\nnsferase,Null mutant is viable; aat1 leu2 double mutant is i\nnviable.\n mYLR243W:Unknown ,, Unknown\n mRPS22B:Homology to rat S15a,ribosomal protein S22B (S24B) (rp50) (Y\nS22),\n mYOL014W:Unknown ,, Unknown\n Cond721:t0-SSD1,M31\n mYNL026W:Unknown ,, Unknown\n mSEC28:Part of a heptameric protein complex that regulates retrogra\nde Golgi-to-ER protein traffic in eukaryotic cells; coatomer\n forms the COP I vesicle coat whose functions are essential,\nepsilon-COP coatomer subunit,Null mutant is viable, shows te\nmperature sensitivity; high copy suppressor of ret1-3\n mYLR427W:Unknown ,, Unknown\n Cond722:t2+SSD1,H44\n mPOM152:May be involved in duplication of nuclear pores and nuclear \npore complexes during S-phase,membrane glycoprotein , nuclea\nr pore complex subunit,Null mutant is viable; overproduction\n inhibits cell growth; synthetically lethal with NUP170 and \nNUP188\n Cond709:t0+Vec\n Cond710:t2+SSD1\n mYGL101W:Unknown ,, Unknown\n
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Computational Genomics Lab, Tel-Aviv uniresity