Module number 1229




Database revision : gnsdb28.10
Date : Tue Feb 25 17:41:43 2003
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mYMR254C:Unknown ,, Unknown\n mTRP1:Note that the sequence of TRP1 from strain S228C, which is t\nhe sequence stored in SGD, contains an ochre mutation at cod\non 67.,N-(5'-phosphoribosyl)-anthranilate isomerase,tryptoph\nan requiring\n mADE5,7:glycinamide ribotide synthetase and aminoimidazole ribotide \nsynthetase,aminoimidazole ribotide synthetase , glycinamide \nribotide synthetase,Adenine requiring\n mYFR024C:Unknown ,, Unknown\n mHAT2:subunit of histone acetyltransferase; may regulate activity \nof Hat1p, the catalytic subunit of histone acetyltransferase\n,histone acetyltransferase subunit,Null mutant is viable\n mATX1:antioxidant protein and metal homeostasis factor, protects a\ngainst oxygen toxicity,copper binding homeostasis protein (p\nutative),hypersensitive toward paraquat (a generator of supe\nroxide anion)\n mFET3:FET3 encodes a ferro-O2-oxidoreductase that is part of the h\nigh-affinity iron transport system,multicopper oxidase,The n\null mutant is viable but defective for high affinity Fe(II) \nuptake. The null mutant is inviable when environmental iron \nis limiting.\n mSEC31:involved in protein transport from endoplasmic reticulum to \nGolgi,COPII coat of secretory pathway vesicles component (p1\n50),Null mutant is inviable\n Cond950:TPK1_MT1val\n Cond954:TPK2_MT2val\n mRAS2:Ras proto-oncogene homolog. Ras2 is involved in growth on no\nn-fermentable carbon sources, the starvation response, sporu\nlation, pseudohyphal growth and aging.,small GTP-binding pro\ntein,Loss of function mutants grow poorly on nonfermentable \ncarbon sources, sporulate in rich media, are unable to diffe\nrentiate into a pseudohyphal form and exhibit an increased l\nife span.\n mMUP1:high affinity methionine permease,high affinity methionine p\nermease,Null mutant is viable but cannot perform high-affini\nty methionine update.\n TPK.TPK:The yeast A kinases differentially regulate iron uptake and \nrespiratory function. Proc Natl Acad Sci U S A. 2000 May 23\n;97(11):5984-8.\n Cond949:WT1val\n mFTR1:Plasma membrane iron permease,iron permease,Lacks high affin\nity iron uptake\n mASN2:Asn1p and Asn2p are isozymes,asparagine synthetase,Null muta\nnt is viable; L-asparagine auxotrophy occurs upon mutation o\nf both ASN1 and ASN2\n SGD.GO:Functional classification via a compendium of knockouts. Hug\nes et.al., cell 2000.\n mSCP160:May be required during cell division for faithful partitioni\nng of the ER-nuclear envelope membranes, involved in control\n of mitotic chromsome transmission,,Null mutant is viable, b\nut exhibits decreased viability, abnormal morphology, and in\ncreased DNA content.\n Cond951:WT2val\n mCAJ1:Homologous to E. coli DnaJ; contains leucine zipper-like mot\nif,,\n Cond955:TPK3_MT1val\n mYCK1:membrane-bound casein kinase I homolog,casein kinase I homol\nog,Null mutant is viable; yck1 yck2 double deletion mutants \nare inviable; yck1 point mutants suppress defective Snf1p ki\nnase activity in snf4 strains\n mOCA1:Unknown ,, Unknown\n mYNL100W:Unknown ,, Unknown\n mSPF1:Sensitivity to a killer toxin (SMK toxin) produced by Pichia\n farinosa,P-type ATPase,The null mutant is viable and resist\nant to the SMK toxin, but grows slowly and has glycosylation\n defects.\n mGDA1:converts nucleoside diphosphates to nucleoside monophosphate\ns to recycle nucleosides and promote transport of additional\n nucleotide sugars into golgi,guanosine diphosphatase of Gol\ngi membrane,Null mutant is viable and has partial block in m\nannosylation of proteins and sphingolipids\n mRIB4:catalyzes synthesis of immediate precursor to riboflavin,6,7\n-dimethyl-8-ribityllumazine synthase (DMRL synthase),Null mu\ntant is viable but is a riboflavin auxotroph\n mBAP2:contains 12 predicted transmembrane domains,amino acid perme\nase for leucine, valine, and isoleucine (putative),reduced u\nptake of leucine, isoleucine, and valine\n mTUP1:general repressor of transcription (with Cyc8p); mediates gl\nucose repression,glucose repression regulatory protein, exhi\nbits similarity to beta subunits of G proteins,Null mutant i\ns viable; exhibits flocculent colony morphology\n Cond952:TPK1_MT2val\n mYGL080W:Unknown ,, Unknown\n mGOT1:Golgi Transport,membrane protein,Null mutant is viable but e\nxhibits ER to Golgi transport defects in vitro. got1 is synt\nhetically lethal with mutations in sft2; the got1 sft2 doubl\ne mutant exhibits defects in transport to the Golgi complex.\n mMSB2:putative integral membrane protein,integral membrane protein\n (putative),multicopy suppressor of cdc24 ts mutation\n Cond956:TPK3_MT2val\n mARN1:Product of gene unknown,,\n mHMX1:Unknown ,, Unknown\n mTAT1:Amino acid transport protein for valine, leucine, isoleucine\n, and tyrosine,amino acid transport protein for valine, leuc\nine, isoleucine, and tyrosine,\n mLEO1:Product of gene unknown,,Null mutant is viable\n mAKL1:Ark-family kinase-like protein. This protein is the third me\nmber (After Ark1p and Prk1p) of the Ark-family kinases in S.\n cerevisiae.,,Null mutant is viable\n mIES5:Ino Eighty Subunit 5,,Null: non essential.\n Cond953:TPK2_MT1val\n mYGR182C:Unknown ,, Unknown\n
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Computational Genomics Lab, Tel-Aviv uniresity